Browsing Departamento de Fisiología y Farmacología by Author "Vidal Sánchez, Verónica"

Now showing items 1-9 of 9

Anti-IL17 treatment ameliorates Down syndrome phenotypes in mice

Rueda Revilla, Noemí; Vidal Sánchez, Verónica; García Cerro, Susana; Narcís Majos, Josep Oriol; Llorens Martín, María; Corrales Pardo, Andrea; Lantigua Romero, Sara; Iglesias, Marcos; Merino Pérez, Jesús; Merino Pérez, Ramón; Martínez-Cué Pesini, Carmen (Elsevier, 2018-10)
Chronic Melatonin Administration Reduced Oxidative Damage and Cellular Senescence in the Hippocampus of a Mouse Model of Down Syndrome

Parisotto, Eduardo B.; Vidal Sánchez, Verónica; García Cerro, Susana; Lantigua Romero, Sara; Filho, Danilo Wilhelm; Sánchez Barceló, Emilio José; Martínez-Cué Pesini, Carmen; Rueda Revilla, Noemí (Springer, 2016)
Chronic melatonin treatment rescues electrophysiological and neuromorphological deficits in a mouse model of Down syndrome

Corrales Pardo, Andrea; Vidal Casado, Rebeca; García Cerro, Susana; Vidal Sánchez, Verónica; Martínez Fernández, Paula; García Iglesias, María Eva; Florez Beledo, Jesús; Sánchez Barceló, Emilio José; Martínez-Cué Pesini, Carmen; Rueda Revilla, Noemí (Blackwell Publishing Ltd, 2014)
Decreasing the Expression of GABAA[alfa]5 Subunit-Containing Receptors Partially Improves Cognitive, Electrophysiological, and Morphological Hippocampal Defects in the Ts65Dn Model of Down Syndrome

Vidal Sánchez, Verónica; García Cerro, Susana; Martínez Fernández, Paula; Corrales Pardo, Andrea; Lantigua Romero, Sara; Vidal Casado, Rebeca; Rueda Revilla, Noemí; Ozmen, Laurence; Hernández, Maria Clemencia; Martínez-Cué Pesini, Carmen (Springer, 2018-06)
Efecto de la reducción de la inhibición mediada por los receptores GABAAα5 y GABAB sobre las alteraciones funcionales y neuromorfológicas en un modelo de síndrome de Down

Vidal Sánchez, Verónica (2017-04-07)
Normalizing the gene dosage of Dyrk1A in a mouse model of Down syndrome rescues several Alzheimer's disease phenotypes

García Cerro, Susana; Rueda Revilla, Noemí; Vidal Sánchez, Verónica; Lantigua Romero, Sara; Martínez-Cué Pesini, Carmen (Elsevier, 2017-06-21)
Overexpression of Dyrk1A Is Implicated in Several Cognitive, Electrophysiological and Neuromorphological Alterations Found in a Mouse Model of Down Syndrome

García Cerro, Susana; Martínez Fernández, Paula; Vidal Sánchez, Verónica; Corrales Pardo, Andrea; Flórez Beledo, Jesús; Vidal Casado, Rebeca; Rueda Revilla, Noemí; Arbonés de Rafael, María Lourdes; Martínez-Cué Pesini, Carmen (Public Library of Science, 2014-09-04)
Pre- and post-natal melatonin administration partially regulates brain oxidative stress but does not improve cognitive or histological alterations in the Ts65Dn mouse model of Down syndrome

Corrales Pardo, Andrea; Parisotto, Eduardo B.; Vidal Sánchez, Verónica; García Cerro, Susana; Lantigua Romero, Sara; Diego, Marian; Filho, Danilo Wilhem; Sánchez Barceló, Emilio José; Martínez-Cué Pesini, Carmen; Rueda Revilla, Noemí (Elsevier, 2017)
Treatment with corn oil improves neurogenesis and cognitive performance in the Ts65Dn mouse model of Down syndrome

Giacomini, Andrea; Stagni, Fiorenza; Emili, Marco; Guidi, Sandra; Salvalai, Maria Elisa; Grilli, Mariagrazia; Vidal Sánchez, Verónica; Martínez-Cué Pesini, Carmen; Bartesaghi, Renata (Elsevier, 2018-06)

Browsing Departamento de Fisiología y Farmacología by Author "Vidal Sánchez, Verónica"

Anti-IL17 treatment ameliorates Down syndrome phenotypes in mice

Chronic Melatonin Administration Reduced Oxidative Damage and Cellular Senescence in the Hippocampus of a Mouse Model of Down Syndrome

Chronic melatonin treatment rescues electrophysiological and neuromorphological deficits in a mouse model of Down syndrome

Decreasing the Expression of GABAA[alfa]5 Subunit-Containing Receptors Partially Improves Cognitive, Electrophysiological, and Morphological Hippocampal Defects in the Ts65Dn Model of Down Syndrome

Efecto de la reducción de la inhibición mediada por los receptores GABAAα5 y GABAB sobre las alteraciones funcionales y neuromorfológicas en un modelo de síndrome de Down

Normalizing the gene dosage of Dyrk1A in a mouse model of Down syndrome rescues several Alzheimer's disease phenotypes

Overexpression of Dyrk1A Is Implicated in Several Cognitive, Electrophysiological and Neuromorphological Alterations Found in a Mouse Model of Down Syndrome

Pre- and post-natal melatonin administration partially regulates brain oxidative stress but does not improve cognitive or histological alterations in the Ts65Dn mouse model of Down syndrome

Treatment with corn oil improves neurogenesis and cognitive performance in the Ts65Dn mouse model of Down syndrome

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Browsing Departamento de Fisiología y Farmacología by Author "Vidal Sánchez, Verónica"

Anti-IL17 treatment ameliorates Down syndrome phenotypes in mice ﻿

Chronic Melatonin Administration Reduced Oxidative Damage and Cellular Senescence in the Hippocampus of a Mouse Model of Down Syndrome ﻿

Chronic melatonin treatment rescues electrophysiological and neuromorphological deficits in a mouse model of Down syndrome ﻿

Decreasing the Expression of GABAA[alfa]5 Subunit-Containing Receptors Partially Improves Cognitive, Electrophysiological, and Morphological Hippocampal Defects in the Ts65Dn Model of Down Syndrome ﻿

Efecto de la reducción de la inhibición mediada por los receptores GABAAα5 y GABAB sobre las alteraciones funcionales y neuromorfológicas en un modelo de síndrome de Down ﻿

Normalizing the gene dosage of Dyrk1A in a mouse model of Down syndrome rescues several Alzheimer's disease phenotypes ﻿

Overexpression of Dyrk1A Is Implicated in Several Cognitive, Electrophysiological and Neuromorphological Alterations Found in a Mouse Model of Down Syndrome ﻿

Pre- and post-natal melatonin administration partially regulates brain oxidative stress but does not improve cognitive or histological alterations in the Ts65Dn mouse model of Down syndrome ﻿

Treatment with corn oil improves neurogenesis and cognitive performance in the Ts65Dn mouse model of Down syndrome ﻿

Anti-IL17 treatment ameliorates Down syndrome phenotypes in mice

Chronic Melatonin Administration Reduced Oxidative Damage and Cellular Senescence in the Hippocampus of a Mouse Model of Down Syndrome

Chronic melatonin treatment rescues electrophysiological and neuromorphological deficits in a mouse model of Down syndrome

Decreasing the Expression of GABAA[alfa]5 Subunit-Containing Receptors Partially Improves Cognitive, Electrophysiological, and Morphological Hippocampal Defects in the Ts65Dn Model of Down Syndrome

Efecto de la reducción de la inhibición mediada por los receptores GABAAα5 y GABAB sobre las alteraciones funcionales y neuromorfológicas en un modelo de síndrome de Down

Normalizing the gene dosage of Dyrk1A in a mouse model of Down syndrome rescues several Alzheimer's disease phenotypes

Overexpression of Dyrk1A Is Implicated in Several Cognitive, Electrophysiological and Neuromorphological Alterations Found in a Mouse Model of Down Syndrome

Pre- and post-natal melatonin administration partially regulates brain oxidative stress but does not improve cognitive or histological alterations in the Ts65Dn mouse model of Down syndrome

Treatment with corn oil improves neurogenesis and cognitive performance in the Ts65Dn mouse model of Down syndrome