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dc.contributor.authorBartesaghi, Renataes_ES
dc.contributor.authorHaydar, Tarik F.es_ES
dc.contributor.authorDelabar, Jean Mauricees_ES
dc.contributor.authorDierssen, Maraes_ES
dc.contributor.authorMartínez-Cué Pesini, Carmen es_ES
dc.contributor.authorBianchi, Diana W.es_ES
dc.contributor.otherUniversidad de Cantabriaes_ES
dc.date.accessioned2017-01-24T17:50:47Z
dc.date.available2017-01-24T17:50:47Z
dc.date.issued2015-10-14es_ES
dc.identifier.issn0270-6474es_ES
dc.identifier.issn1529-2401es_ES
dc.identifier.urihttp://hdl.handle.net/10902/10133
dc.description.abstractDown syndrome (DS) is a relatively common genetic condition caused by the triplication of human chromosome 21. No therapies currently exist for the rescue of neurocognitive impairment in DS. This review presents exciting findings showing that it is possible to restore brain development and cognitive performance in mouse models of DS with therapies that can also apply to humans. This knowledge provides a potential breakthrough for the prevention of intellectual disability in DS.es_ES
dc.format.extent10 p.es_ES
dc.language.isoenges_ES
dc.publisherSociety for Neurosciencees_ES
dc.rights© Society for Neurosciencees_ES
dc.sourceJournal of neuroscience, October 14, 2015; 35(41): 13843-13852es_ES
dc.titleNew Perspectives for the Rescue of Cognitive Disability in Down Syndromees_ES
dc.typeinfo:eu-repo/semantics/articlees_ES
dc.relation.publisherVersionhttp://www.jneurosci.org/content/35/41/13843.longes_ES
dc.rights.accessRightsopenAccesses_ES
dc.identifier.DOI10.1523/JNEUROSCI.2775-15.2015es_ES
dc.type.versionpublishedVersiones_ES


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