Browsing by UC author "Corrales Pardo, Andrea"

Now showing items 1-6 of 6

Anti-IL17 treatment ameliorates Down syndrome phenotypes in mice

Rueda Revilla, Noemí; Vidal Sánchez, Verónica; García Cerro, Susana; Narcís Majos, Josep Oriol; Llorens Martín, María; Corrales Pardo, Andrea; Lantigua Romero, Sara; Iglesias, Marcos; Merino Pérez, Jesús; Merino Pérez, Ramón; Martínez-Cué Pesini, Carmen (Elsevier, 2018-10)
Chronic melatonin treatment rescues electrophysiological and neuromorphological deficits in a mouse model of Down syndrome

Corrales Pardo, Andrea; Vidal Casado, Rebeca; García Cerro, Susana; Vidal Sánchez, Verónica; Martínez Fernández, Paula; García Iglesias, María Eva; Florez Beledo, Jesús; Sánchez Barceló, Emilio José; Martínez-Cué Pesini, Carmen; Rueda Revilla, Noemí (Blackwell Publishing Ltd, 2014)
Decreasing the Expression of GABAA[alfa]5 Subunit-Containing Receptors Partially Improves Cognitive, Electrophysiological, and Morphological Hippocampal Defects in the Ts65Dn Model of Down Syndrome

Vidal Sánchez, Verónica; García Cerro, Susana; Martínez Fernández, Paula; Corrales Pardo, Andrea; Lantigua Romero, Sara; Vidal Casado, Rebeca; Rueda Revilla, Noemí; Ozmen, Laurence; Hernández, Maria Clemencia; Martínez-Cué Pesini, Carmen (Springer, 2018-06)
Estudio de los efectos protectores del tratamiento crónico con melatonina sobre los déficits cognitivos del ratón Ts65Dn : un modelo de síndrome de Down

Corrales Pardo, Andrea (2015-10-16)
Overexpression of Dyrk1A Is Implicated in Several Cognitive, Electrophysiological and Neuromorphological Alterations Found in a Mouse Model of Down Syndrome

García Cerro, Susana; Martínez Fernández, Paula; Vidal Sánchez, Verónica; Corrales Pardo, Andrea; Flórez Beledo, Jesús; Vidal Casado, Rebeca; Rueda Revilla, Noemí; Arbonés de Rafael, María Lourdes; Martínez-Cué Pesini, Carmen (Public Library of Science, 2014-09-04)
Pre- and post-natal melatonin administration partially regulates brain oxidative stress but does not improve cognitive or histological alterations in the Ts65Dn mouse model of Down syndrome

Corrales Pardo, Andrea; Parisotto, Eduardo B.; Vidal Sánchez, Verónica; García Cerro, Susana; Lantigua Romero, Sara; Diego, Marian; Filho, Danilo Wilhem; Sánchez Barceló, Emilio José; Martínez-Cué Pesini, Carmen; Rueda Revilla, Noemí (Elsevier, 2017)

Browsing by UC author "Corrales Pardo, Andrea"

Anti-IL17 treatment ameliorates Down syndrome phenotypes in mice

Chronic melatonin treatment rescues electrophysiological and neuromorphological deficits in a mouse model of Down syndrome

Decreasing the Expression of GABAA[alfa]5 Subunit-Containing Receptors Partially Improves Cognitive, Electrophysiological, and Morphological Hippocampal Defects in the Ts65Dn Model of Down Syndrome

Estudio de los efectos protectores del tratamiento crónico con melatonina sobre los déficits cognitivos del ratón Ts65Dn : un modelo de síndrome de Down

Overexpression of Dyrk1A Is Implicated in Several Cognitive, Electrophysiological and Neuromorphological Alterations Found in a Mouse Model of Down Syndrome

Pre- and post-natal melatonin administration partially regulates brain oxidative stress but does not improve cognitive or histological alterations in the Ts65Dn mouse model of Down syndrome

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Browsing by UC author "Corrales Pardo, Andrea"

Anti-IL17 treatment ameliorates Down syndrome phenotypes in mice ﻿

Chronic melatonin treatment rescues electrophysiological and neuromorphological deficits in a mouse model of Down syndrome ﻿

Decreasing the Expression of GABAA[alfa]5 Subunit-Containing Receptors Partially Improves Cognitive, Electrophysiological, and Morphological Hippocampal Defects in the Ts65Dn Model of Down Syndrome ﻿

Estudio de los efectos protectores del tratamiento crónico con melatonina sobre los déficits cognitivos del ratón Ts65Dn : un modelo de síndrome de Down ﻿

Overexpression of Dyrk1A Is Implicated in Several Cognitive, Electrophysiological and Neuromorphological Alterations Found in a Mouse Model of Down Syndrome ﻿

Pre- and post-natal melatonin administration partially regulates brain oxidative stress but does not improve cognitive or histological alterations in the Ts65Dn mouse model of Down syndrome ﻿

Anti-IL17 treatment ameliorates Down syndrome phenotypes in mice

Chronic melatonin treatment rescues electrophysiological and neuromorphological deficits in a mouse model of Down syndrome

Decreasing the Expression of GABAA[alfa]5 Subunit-Containing Receptors Partially Improves Cognitive, Electrophysiological, and Morphological Hippocampal Defects in the Ts65Dn Model of Down Syndrome

Estudio de los efectos protectores del tratamiento crónico con melatonina sobre los déficits cognitivos del ratón Ts65Dn : un modelo de síndrome de Down

Overexpression of Dyrk1A Is Implicated in Several Cognitive, Electrophysiological and Neuromorphological Alterations Found in a Mouse Model of Down Syndrome

Pre- and post-natal melatonin administration partially regulates brain oxidative stress but does not improve cognitive or histological alterations in the Ts65Dn mouse model of Down syndrome