xmlui.themes.about.link
Instituto de Biomedicina y Biotecnología de Cantabria (IBBTEC): Envíos recientes
Mostrando ítems 201-220 de 221
-
MYC as therapeutic target in leukemia and lymphoma
(Dove Medical Press, 2015-07-22) -
Schizophrenia gene expression profile reverted to normal levels by antipsychotics
(Oxford University Press, 2014-10-31) -
Synthetic Fatty Acids Prevent Plasmid-Mediated Horizontal Gene Transfer
(American Society for Microbiology, 2015-09-01) -
Spatial control of Cdc42 signalling by a GM130-RasGRF complex regulates polarity and tumorigenesis
(Nature Publishing Group, 2014-09-11) -
Endocytosis as a biological response in receptor pharmacology: evaluation by fluorescence microscopy
(Public Library of Science, 2015-04-07) -
Revisiting the role of raphe and serotonin in neuropsychiatric disorders
(Rockefeller University Press, 2015-04) -
Colorectal adenomas contain multiple somatic mutations that do not coincide with synchronous adenocarcinoma specimens
(Public Library of Science, 2015-03-16) -
Recurrent chromosomal gains and heterogeneous driver mutations characterise papillary renal cancer evolution
(Nature Publishing Group, 2015-03-19) -
Leukemia-associated somatic mutations drive distinct patterns of age-related clonal hemopoiesis
(Elsevier, 2015-03-03) -
Two novel type 2 diabetes loci revealed through integration of TCF7L2 DNA occupancy and SNP association data
(BMJ Publishing Group, 2014-12-01) -
A high security double lock and key mechanism in HUH relaxases controls oriT-processing for plasmid conjugation
(Oxford University Press, 2014-08-14) -
Key components of the eight classes of type IV secretion systems involved in bacterial conjugation or protein secretion
(Oxford University Press, 2014-05-01) -
Another tool in the genome-wide association study arsenal: population-based detection of somatic gene conversion
(BioMed Central, 2011-02-03) -
Fast regulation of AP-1 activity through interaction of lamin A/C, ERK1/2, and c-Fos at the nuclear envelope
(Rockefeller University Press, 2008-11-17) -
ERK1/2 MAP kinases promote cell cycle entry by rapid, kinase-independent disruption of retinoblastoma-lamin A complexes
(Rockefeller University Press, 2010-11-29)