Visualizar por autor "Lantigua Romero, Sara"

Anti-IL17 treatment ameliorates Down syndrome phenotypes in mice

Rueda Revilla, Noemí

; Vidal Sánchez, Verónica

; García Cerro, Susana

; Narcís Majos, Josep Oriol; Llorens Martín, María; Corrales Pardo, Andrea

; Lantigua Romero, Sara

; Iglesias, Marcos; Merino Pérez, Jesús

; Merino Pérez, Ramón

; Martínez-Cué, Carmen

(Elsevier, 2018-10)

Cerebellar alterations in a model of Down syndrome: The role of the Dyrk1A gene

García Cerro, Susana

; Vidal Sánchez, Verónica

; Lantigua Romero, Sara

; Berciano Blanco, María Teresa

; Lafarga Coscojuela, Miguel Ángel

; Ramos Cabrer, Pedro; Padro, Daniel; Rueda Revilla, Noemí

; Martínez-Cué, Carmen

(Elsevier, 2017-12)

Chronic Melatonin Administration Reduced Oxidative Damage and Cellular Senescence in the Hippocampus of a Mouse Model of Down Syndrome

Parisotto, Eduardo B.; Vidal Sánchez, Verónica

; García Cerro, Susana

; Lantigua Romero, Sara

; Filho, Danilo Wilhelm; Sánchez Barceló, Emilio José

; Martínez-Cué, Carmen

; Rueda Revilla, Noemí

(Springer, 2016)

Decreasing the Expression of GABAA[alfa]5 Subunit-Containing Receptors Partially Improves Cognitive, Electrophysiological, and Morphological Hippocampal Defects in the Ts65Dn Model of Down Syndrome

Vidal Sánchez, Verónica

; García Cerro, Susana

; Martínez Fernández, Paula

; Corrales Pardo, Andrea

; Lantigua Romero, Sara

; Vidal Casado, Rebeca; Rueda Revilla, Noemí

; Ozmen, Laurence; Hernández, Maria Clemencia; Martínez-Cué, Carmen

(Springer, 2018-06)

Efectos de distintas estrategias terapéuticas sobre los déficits cognitivos y las alteraciones neuromorfológicas de un modelo de síndrome de Down

Lantigua Romero, Sara

(2017-07-14)

Normalizing the gene dosage of Dyrk1A in a mouse model of Down syndrome rescues several Alzheimer's disease phenotypes

García Cerro, Susana

; Rueda Revilla, Noemí

; Vidal Sánchez, Verónica

; Lantigua Romero, Sara

; Martínez-Cué, Carmen

(Elsevier, 2017-06-21)

Pre- and post-natal melatonin administration partially regulates brain oxidative stress but does not improve cognitive or histological alterations in the Ts65Dn mouse model of Down syndrome

Corrales Pardo, Andrea

; Parisotto, Eduardo B.; Vidal Sánchez, Verónica

; García Cerro, Susana

; Lantigua Romero, Sara

; Diego, Marian; Filho, Danilo Wilhem; Sánchez Barceló, Emilio José

; Martínez-Cué, Carmen

; Rueda Revilla, Noemí

(Elsevier, 2017)

Prenatal Administration of Oleic Acid or Linolenic Acid Reduces Neuromorphological and Cognitive Alterations in Ts65dn Down Syndrome Mice

García Cerro, Susana

; Rueda Revilla, Noemí

; Vidal Sánchez, Verónica

; Puente Bedia, Alba

; Campa Fernández, Víctor Manuel; Lantigua Romero, Sara

; Narcís Majos, Josep Oriol; Velasco, Ana; Bartesaghi, Renata; Martínez-Cué, Carmen

(Oxford University Press, 2020-06-01)

Prenatal, but not Postnatal, Curcumin Administration Rescues Neuromorphological and Cognitive Alterations in Ts65Dn Down Syndrome Mice

Rueda Revilla, Noemí

; Vidal Sánchez, Verónica

; García Cerro, Susana

; Puente Bedia, Alba

; Campa Fernández, Víctor Manuel; Lantigua Romero, Sara

; Narcís Majos, Josep Oriol; Bartesaghi, Renata; Martínez-Cué, Carmen

(Oxford University Press, 2020-09-01)

Visualizar por autor "Lantigua Romero, Sara"

Anti-IL17 treatment ameliorates Down syndrome phenotypes in mice

Cerebellar alterations in a model of Down syndrome: The role of the Dyrk1A gene

Chronic Melatonin Administration Reduced Oxidative Damage and Cellular Senescence in the Hippocampus of a Mouse Model of Down Syndrome

Decreasing the Expression of GABAA[alfa]5 Subunit-Containing Receptors Partially Improves Cognitive, Electrophysiological, and Morphological Hippocampal Defects in the Ts65Dn Model of Down Syndrome

Efectos de distintas estrategias terapéuticas sobre los déficits cognitivos y las alteraciones neuromorfológicas de un modelo de síndrome de Down

Normalizing the gene dosage of Dyrk1A in a mouse model of Down syndrome rescues several Alzheimer's disease phenotypes

Pre- and post-natal melatonin administration partially regulates brain oxidative stress but does not improve cognitive or histological alterations in the Ts65Dn mouse model of Down syndrome

Prenatal Administration of Oleic Acid or Linolenic Acid Reduces Neuromorphological and Cognitive Alterations in Ts65dn Down Syndrome Mice

Prenatal, but not Postnatal, Curcumin Administration Rescues Neuromorphological and Cognitive Alterations in Ts65Dn Down Syndrome Mice

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Visualizar por autor "Lantigua Romero, Sara"

Anti-IL17 treatment ameliorates Down syndrome phenotypes in mice ﻿

Cerebellar alterations in a model of Down syndrome: The role of the Dyrk1A gene ﻿

Chronic Melatonin Administration Reduced Oxidative Damage and Cellular Senescence in the Hippocampus of a Mouse Model of Down Syndrome ﻿

Decreasing the Expression of GABAA[alfa]5 Subunit-Containing Receptors Partially Improves Cognitive, Electrophysiological, and Morphological Hippocampal Defects in the Ts65Dn Model of Down Syndrome ﻿

Efectos de distintas estrategias terapéuticas sobre los déficits cognitivos y las alteraciones neuromorfológicas de un modelo de síndrome de Down ﻿

Normalizing the gene dosage of Dyrk1A in a mouse model of Down syndrome rescues several Alzheimer's disease phenotypes ﻿

Pre- and post-natal melatonin administration partially regulates brain oxidative stress but does not improve cognitive or histological alterations in the Ts65Dn mouse model of Down syndrome ﻿

Prenatal Administration of Oleic Acid or Linolenic Acid Reduces Neuromorphological and Cognitive Alterations in Ts65dn Down Syndrome Mice ﻿

Prenatal, but not Postnatal, Curcumin Administration Rescues Neuromorphological and Cognitive Alterations in Ts65Dn Down Syndrome Mice ﻿

Anti-IL17 treatment ameliorates Down syndrome phenotypes in mice

Cerebellar alterations in a model of Down syndrome: The role of the Dyrk1A gene

Chronic Melatonin Administration Reduced Oxidative Damage and Cellular Senescence in the Hippocampus of a Mouse Model of Down Syndrome

Decreasing the Expression of GABAA[alfa]5 Subunit-Containing Receptors Partially Improves Cognitive, Electrophysiological, and Morphological Hippocampal Defects in the Ts65Dn Model of Down Syndrome

Efectos de distintas estrategias terapéuticas sobre los déficits cognitivos y las alteraciones neuromorfológicas de un modelo de síndrome de Down

Normalizing the gene dosage of Dyrk1A in a mouse model of Down syndrome rescues several Alzheimer's disease phenotypes

Pre- and post-natal melatonin administration partially regulates brain oxidative stress but does not improve cognitive or histological alterations in the Ts65Dn mouse model of Down syndrome

Prenatal Administration of Oleic Acid or Linolenic Acid Reduces Neuromorphological and Cognitive Alterations in Ts65dn Down Syndrome Mice

Prenatal, but not Postnatal, Curcumin Administration Rescues Neuromorphological and Cognitive Alterations in Ts65Dn Down Syndrome Mice