@article{10902/31358,
year = {2013},
url = {https://hdl.handle.net/10902/31358},
abstract = {Insulin resistance is an essential feature of the metabolic syndrome that has been linked to rheumatoid arthritis (RA). Understanding how inflammation arising in one tissue affects the physiology and pathology of other organs remains an unanswered question with therapeutic implications for chronic conditions including obesity, diabetes mellitus, atherosclerosis, and RA. Adipokines may play a role in the development of atherogenesis in patients with RA. Biologic therapies, such as TNF-∝ antagonists, that block proinflammatory cytokines have beneficial effects on the insulin resistance that is often observed in patients with RA.},
organization = {This study was supported by grants from “Fondo de Investigaciones Sanitarias” PI06/0024, PS09/00748, and PI12/00060 (Spain). This work was also partially supported by RETICS Program, RD08/0075 (RIER) from “Instituto de Salud Carlos III” (ISCIII) (Spain). R. L.-M. is a recipient of a Sara Borrell postdoctoral fellowship from the Instituto Carlos III de Salud at the Spanish Ministry of Health.},
publisher = {Hindawi Publishing Corporation},
publisher = {Mediators of Inflammation, 2013, 710928},
title = {Metabolic syndrome in rheumatoid arthritis},
author = {Ferraz-Amaro, Iván and González Juanatey, Carlos and López Mejías, Raquel and Riancho Zarrabeitia, Leyre and González-Gay Mantecón, Miguel Ángel},
}